A research group of the Japan Zoological Research Institute said at an international conference in Sendai City on the 1st that the team found a very small amount of mutated saw protein in peripheral nerves and paraneins outside the specific danger zone of cattle infected with mad cow disease. It was the first time in the world that the mutated saw protein associated with mad cow disease was discovered. The mad cow disease and the new Creutzfeldt-Jakob disease caused by humans are all caused by variation in sawn protein. According to a report from Kyodo News Agency on the 2nd, the Japanese scientific community generally believes that the brain and spinal cord of cattle are the specific dangerous sites for accumulating mutated saw proteins. As a precautionary measure, Japan has stipulated that meat processing plants are obliged to remove the brain and spinal cord of cattle, and that the peripheral nerves and an organ that covers the kidneys are always considered to be "missing" from variogenous sawn proteins. However, the research team led by Shinichi Shinichi from the 11th mad cow in Japan found in March 2004 this year extracted samples containing peripheral nerves such as pararenal tissue, sciatic nerve, and tibial nerve and tested positive for them. reaction. Subsequent analysis showed that there was a very small amount of variant sawing protein in the peripheral nerves and paraneumons examined. In the future, the research team will also examine other parts of the mad cow. The Japanese Ministry of Health, Labour and Welfare has also stated that it will conduct an infection experiment and the Japanese Food Safety Committee will decide whether or not to redefine the specific risk areas of cows suffering from mad cow disease. The University of Tokyo professor Onodera believes that the mad cow disease in the 11th mad cow discovered in Japan is in late stages. It is not unbelievable that mutated sawnin is spread to its peripheral nerves, and the amount of mutated saw protein detected is very small, from the perspective of food safety. Look, it may not be necessary to set peripheral nerves and other tissues and organs as specific dangerous sites.
The level of IgM antibody begins to rise after 1 week after the initial infection, while the IgG appears later than IgM (usually in 14 days after infection) and can last for 6 months or even several years, which meansthat the IgG serves as an indicator of previous infection. Suspected patients that are infected by 2019-nCoV can be rapidly identified by simultaneous monitoring of IgM and IgG. During the outbreak period of2003-SARS and the 2016-Zika, IgM / IgG antibody detection was used as one of the recommendeddiagnostic methods.
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