Chronic respiratory disease in chickens is a chronic respiratory disease in chickens. The pathogen is chicken pleuritis microorganisms and is a septic mycoplasma. In particular, layer chickens from 90 days to 300 days old and chicks from 40 days to 50 days old are most susceptible. Diseased chickens and diseased chickens are the source of infection of the disease. The disease is mainly transmitted through the respiratory tract. Infection through eggs is the main reason for the transmission of this disease from generation to generation. Local annual prosperity in the winter and spring seasons greatly affects the production efficiency of the chicken industry.
Incidence: On March 16, 2012, 4,500 280-day layer chickens raised at a farm in Chao Town, Chaohu City, Anhui province developed respiratory symptoms and the egg production rate decreased by 5% to 10%. The auscultation chickens had dry rales. Individuals with eyelid edema and conjunctivitis were initially diagnosed as chronic respiratory disease in chickens. Tiamulin and roxithromycin were used to treat one course of treatment. The respiratory symptoms of chickens disappeared and the egg production rate gradually returned to normal levels. The chickens gradually recovered.
Clinical symptoms The incubation period of this disease is generally 4 days to 7 days. The loss of appetite at the beginning of the disease reduces the egg production rate. The nasal cavity flows out of the slurry, and some cough and sneeze. When the cough occurs, dry rales occur. The diseased chicken often shakes his head to excrete nasal exudates. In the presence of rhinitis, inflammation of the mouth, pharynx, larynx, and trachea also occurred. In some cases, the tongue, tongue, or cheeks are covered with mucous membranes with large or small needles, large grayish white or yellow-white throat-like membranes; the exudates in the larynx and trachea are changed from serous to mucous, cheese-like, and blocked. Throat and trachea, so when the inspiratory disease chickens often open their mouths to stretch their necks, when the breath is narrowed neck, and intermittently issued a squeak, quacking call, if not treated, often due to suffocation. In addition, one-sided conjunctivitis occurs in the eye, and when the disease is severe, the vision of the diseased chicken is diminished or blind. Most diseased chickens develop diarrhea at the end of the disease.
Pathological changes mainly in the respiratory organs, which have a characteristic change in the airbag. The airbag membrane is turbid, thickened, and has reduced transparency. There are yellow-white bean dregs-like exudates, and there are often large nodules from sesame to soybeans. In addition, there was increased mucus in the nasal cavity and trachea, mild edema of the tracheal mucosa, and annular bleeding of the trachea. In a few cases, salpingitis, mild pericarditis, and pericarditis occur, from which E. coli can be isolated.
Diagnosis can be based on the characteristics of the disease, clinical symptoms, pathological changes and airway inflammation and other characteristic pathological changes, can make a preliminary diagnosis. Diagnosis requires the necessary laboratory diagnosis. The differential diagnosis focuses on the differences between infectious bronchitis, infectious rhinitis, mucous fowl pox, and atypical Newcastle disease.
The difference between infectious bronchitis and infectious bronchitis is rapid, and antibacterial treatment is less effective. The majority of chicken farms are also immunized with H120 and H52. The incidence of infectious bronchitis is relatively small. Generally can be ruled out.
Differentiation from infectious rhinitis Infectious rhinitis has a rapid onset, generally spreading from 3 days to 5 days. The egg production rate after laying hens is reduced by 10% to 30%. It takes about 20 days before the egg production rate starts to rise. Less than the original level. Chickens with infectious rhinitis have severe facial edema, while only a few chickens with chronic respiratory diseases are swollen and slightly facial.
Differentiation from mucous fowl pox Type mucous fowlpox forms yellow-white nodules on the oral cavity, throat, and tracheal mucosa. Small nodules fuse together to form a yellow-white, cheese-like pseudomembrane that is not easily peeled off. Sometimes it also affects the skin of the eye mucous membranes and other parts of the body and is easy to distinguish from this disease.
Differences from Atypical Newcastle Disease When atypical Newcastle disease occurs, it is common for some chickens to have a subacute onset, yellowish greenish dilute feces, and chicken crown clamps. The autopsy revealed glandular papillary and cloacal mucosa with hemorrhagic lesions, small intestine mucosa with island-like necrotic lesions, and the diseased chicken breathed out with a wet rales that could be discerned with the disease.
Comprehensive prevention and control methods 1, usually strengthen the feeding and management, improve the health conditions, reduce the breeding density, the chicken house to be well ventilated, fresh air, suitable temperature. Once onset, timely treatment, strict disinfection.
2. According to the actual situation of the farm (household) and the incidence of chronic respiratory diseases in chickens in this area, a reasonable immunization program can be formulated. The vaccine can be domestically or imported “F-milk mycoplasma attenuated vaccine†at 7 days of age. 14-day eye drops are better.
3, drug treatment: a large group of mixed use of tiamulin mixed drink, 100 grams of water 200 liters, once every 3 days to 5 days; then use roxithromycin mixed drink, 1000 liters per 100 grams of water, use 3 days ~ 5 days.
4, the incidence of late and late stage of disease can be used for heavy chicken, astragalus polysaccharide injection 100 ml plus 1 million international units of streptomycin 2, 800,000 international units of penicillin 2 and dubbed mixed solution, 0.5 ml per kg body weight injection, For 3 consecutive days, there is a good effect.
Incidence: On March 16, 2012, 4,500 280-day layer chickens raised at a farm in Chao Town, Chaohu City, Anhui province developed respiratory symptoms and the egg production rate decreased by 5% to 10%. The auscultation chickens had dry rales. Individuals with eyelid edema and conjunctivitis were initially diagnosed as chronic respiratory disease in chickens. Tiamulin and roxithromycin were used to treat one course of treatment. The respiratory symptoms of chickens disappeared and the egg production rate gradually returned to normal levels. The chickens gradually recovered.
Clinical symptoms The incubation period of this disease is generally 4 days to 7 days. The loss of appetite at the beginning of the disease reduces the egg production rate. The nasal cavity flows out of the slurry, and some cough and sneeze. When the cough occurs, dry rales occur. The diseased chicken often shakes his head to excrete nasal exudates. In the presence of rhinitis, inflammation of the mouth, pharynx, larynx, and trachea also occurred. In some cases, the tongue, tongue, or cheeks are covered with mucous membranes with large or small needles, large grayish white or yellow-white throat-like membranes; the exudates in the larynx and trachea are changed from serous to mucous, cheese-like, and blocked. Throat and trachea, so when the inspiratory disease chickens often open their mouths to stretch their necks, when the breath is narrowed neck, and intermittently issued a squeak, quacking call, if not treated, often due to suffocation. In addition, one-sided conjunctivitis occurs in the eye, and when the disease is severe, the vision of the diseased chicken is diminished or blind. Most diseased chickens develop diarrhea at the end of the disease.
Pathological changes mainly in the respiratory organs, which have a characteristic change in the airbag. The airbag membrane is turbid, thickened, and has reduced transparency. There are yellow-white bean dregs-like exudates, and there are often large nodules from sesame to soybeans. In addition, there was increased mucus in the nasal cavity and trachea, mild edema of the tracheal mucosa, and annular bleeding of the trachea. In a few cases, salpingitis, mild pericarditis, and pericarditis occur, from which E. coli can be isolated.
Diagnosis can be based on the characteristics of the disease, clinical symptoms, pathological changes and airway inflammation and other characteristic pathological changes, can make a preliminary diagnosis. Diagnosis requires the necessary laboratory diagnosis. The differential diagnosis focuses on the differences between infectious bronchitis, infectious rhinitis, mucous fowl pox, and atypical Newcastle disease.
The difference between infectious bronchitis and infectious bronchitis is rapid, and antibacterial treatment is less effective. The majority of chicken farms are also immunized with H120 and H52. The incidence of infectious bronchitis is relatively small. Generally can be ruled out.
Differentiation from infectious rhinitis Infectious rhinitis has a rapid onset, generally spreading from 3 days to 5 days. The egg production rate after laying hens is reduced by 10% to 30%. It takes about 20 days before the egg production rate starts to rise. Less than the original level. Chickens with infectious rhinitis have severe facial edema, while only a few chickens with chronic respiratory diseases are swollen and slightly facial.
Differentiation from mucous fowl pox Type mucous fowlpox forms yellow-white nodules on the oral cavity, throat, and tracheal mucosa. Small nodules fuse together to form a yellow-white, cheese-like pseudomembrane that is not easily peeled off. Sometimes it also affects the skin of the eye mucous membranes and other parts of the body and is easy to distinguish from this disease.
Differences from Atypical Newcastle Disease When atypical Newcastle disease occurs, it is common for some chickens to have a subacute onset, yellowish greenish dilute feces, and chicken crown clamps. The autopsy revealed glandular papillary and cloacal mucosa with hemorrhagic lesions, small intestine mucosa with island-like necrotic lesions, and the diseased chicken breathed out with a wet rales that could be discerned with the disease.
Comprehensive prevention and control methods 1, usually strengthen the feeding and management, improve the health conditions, reduce the breeding density, the chicken house to be well ventilated, fresh air, suitable temperature. Once onset, timely treatment, strict disinfection.
2. According to the actual situation of the farm (household) and the incidence of chronic respiratory diseases in chickens in this area, a reasonable immunization program can be formulated. The vaccine can be domestically or imported “F-milk mycoplasma attenuated vaccine†at 7 days of age. 14-day eye drops are better.
3, drug treatment: a large group of mixed use of tiamulin mixed drink, 100 grams of water 200 liters, once every 3 days to 5 days; then use roxithromycin mixed drink, 1000 liters per 100 grams of water, use 3 days ~ 5 days.
4, the incidence of late and late stage of disease can be used for heavy chicken, astragalus polysaccharide injection 100 ml plus 1 million international units of streptomycin 2, 800,000 international units of penicillin 2 and dubbed mixed solution, 0.5 ml per kg body weight injection, For 3 consecutive days, there is a good effect.
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